Indian Journal of Chest Diseases & Allied Sciences
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Item A 12-year-old girl with hodgkin’s disease presenting as a non-resolving pneumonia and an endobronchial nodule.(2009-10) Pinto, Lancelot Mark; Udwadia, Zarir FarokhThe presence of a non-resolving pneumonia warrants the suspicion of a possible malignancy. While pulmonary involvement in Hodgkin’s disease can present as a non-resolving pneumonia, the clinical clues of dyspnoea, stridor and wheeze point to a possible endobronchial involvement. A bronchoscopy in such a situation can be valuable for diagnosis, and can aid in staging of the disease. The true incidence of endobronchial involvement in Hodgkin’s disease is not known, but when diagnosed early and treated appropriately, the prognosis is usually good, and a complete cure is possible.Item A 54-Year-Old Man with Tracheomegaly, Tracheal Diverticulas and Bronchiectasis — Mounier-Kuhn Syndrome.(2015-04) Singh, Mandeep; Kumar, RajMounier-Kuhn syndrome is a rare idiopathic clinical, radiological and bronchoscopic disorder characterised by abnormal dilatation of the tracheo-bronchial tree. The usual presentation is with recurrent lower respiratory tract infections. Herein, we report the case of an adult male who was diagnosed to have Mounier-Kuhn syndrome based on radiographic finding of a tracheal diameter of 45.5 mm on computed tomography and dynamic complete collapse of the tracheo-bronchial tree on forced expiration, observed during ronchoscopy.Item Aberrant right subclavian artery causing dysphagia.(1981-10-01) Mohan, V; Raizada, R M; Hasan, W; Khushu, RItem Abnormalities of lipid metabolism in asthma and rhinitis--a comprehensive study.(1987-07-01) Wadehra, N R; Chhabra, S K; Gaur, S N; Joshi, A P; Agrawal, K PItem Abstracts’ Service.(2015-01) The Indian Journal of Chest Diseases and Allied SciencesItem Accidental condom inhalation.(2004-01-12) Arya, C L; Gupta, Rajnish; Arora, V KA 27-year-old lady presented with persistent cough, sputum and fever for the preceding six months. Inspite of trials with antibiotics and anti-tuberculosis treatment for the preceeding four months, her symptoms did not improve. A subsequent chest radiograph showed non-homogeneous collapse-consolidation of right upper lobe. Videobronchoscopy revealed an inverted bag like structure in right upper lobe bronchus and rigid bronchoscopic removal with biopsy forceps confirmed the presence of a condom. Detailed retrospective history also confirmed accidental inhalation of the condom during fellatio.Item Achalasia cardia: uncommon presentation.(2001-04-01) Mohan, A; Ibrarullah, M; Sonawane, R; Dilip, D; Chandra, A; Sharma, S KItem Achalasia of the oesophagus.(1983-07-01) Razaque, M A; Singh, L S; Singh, N TItem Acromesomelic dysplasia with bronchiectasis.(2005-04-19) Farnaz, Sarwat; Gothi, D; Joshi, J MWe report a rare case of acromesomelic dysplasia with bilateral bronchiectasis and obstructive sleep apnoea. Diagnosis of acromesomelic dysplasia was based on radiographs of whole skeleton.Item Actinomycosis of the lung.(1978-01-01) Narang, R K; Dubey, A LItem Acute accidental exposure to chlorine fumes--a study of 82 cases.(1992-04-01) Moulick, N D; Banavali, S; Abhyankar, A D; Borkar, S; Aiyengar, J; Kapadia, N M; Khokhani, R CEighty-two patients were hospitalized following an accidental exposure to chlorine. All patients presented with dyspnoea and cough. The other symptoms included irritation of throat (53.6%), irritation of eyes (42.3%), headache (29.2%), abdominal pain (26.8%), vomiting (24.3%) and giddiness (9.7%). All of them had bronchospasm and 5 (6%) had cyanosis at the onset. An x-ray of the chest revealed patchy infiltrates in 3 (3.85%) and hilar congestion in 2 (2.44%). Pulmonary function tests showed an obstructive pattern in 27.4%, restrictive in 3.25% and mixed in 53.2%. Pulmonary functions were normal in 16.1% of the patients. Bronchoscopy revealed tracheobronchial mucosal congestion in all cases, hemorrhagic spots in 35.7%, erosions and ulcers in 12.5%. All patients were treated with oxygen, aminophylline, hydrocortisone and antibiotics. Haematemesis (n = 1) and pulmonary oedema (n = 2) developed 12 hours after the admission. Two other patients developed pneumonia 48 hours later. All patients recovered satisfactorily. On follow-up 16 patients had no sequelae after one year. Pulmonary functions were normal in 5 patients after 3 years of follow-up.Item Acute accidental exposure to chlorine gas: Clinical presentation, pulmonary functions and outcomes.(2010-07) Mohan, Alladi; Kumar, S Naveen; Rao, M H; Bollineni, S; Manohar, I ChiranjeeviBackground. To study the clinical presentation, pulmonary functions and outcomes in subjects who were accidentally exposed to chlorine gas. Methods. Prospective observational study of 64 patients who sustained acute accidental exposure to chlorine gas during a leak in the chlorination system of the public bathing pool of a temple. Results. The major presenting symptoms and signs included acute dyspnoea (100%), chest discomfort (100%), cough (97%), eye irritation (88%), giddiness (72%), vomiting (46%), and heaviness in the head (44%); tachycardia (100%), tachypnoea (96%) and polyphonic wheezing (28%). All patients were managed in the emergency room with humidified oxygen inhalation and beta-2 agonist nebulisation and 52 were discharged within six hours. Twelve patients were severely affected and required hospitalisation; three of them were admitted into the intensive care unit. Three patients developed pulmonary oedema six to eight hours following admission. Pulmonary function testing (n=12) at presentation revealed obstructive defect in eight and mixed obstructive-cum-restrictive defect in four patients. The mean duration of hospital stay was 5.1±2.1 days. None of the patients died. Reactive airway dysfunction syndrome (RADS) was observed in three of the 12 hospitalised patients, who complained of manifested persistent cough that lasted for three months period following discharge. Serial pulmonary functions recovered to normal range by the end of the six months in all patients and remained so at one-year follow-up. Conclusion. Acute exposure to chlorine gas is an uncommon, but important public health hazard and can cause RADS, acute lung injury and pulmonary function abnormalities , which are reversible on prompt and appropriate management.Item Acute antiarrhythmic effects of intravenous verapamil (Isoptin) in supraventricular tachy-arrhythmias.(1977-04-01) Kuruvilla, A; Subramaniam, N; Abraham, K A; Stephen, P; Cherian, GItem Acute aortic regurgitation.(1979-10-01) Mittal, S R; Wahi, P LItem Acute chest syndrome in adult homozygous sickle cell disease.(1986-01-01) Jain, A P; Chaubey, B SItem Acute effects of tobacco inhalation in the form of 'snuff' on cardio-respiratory parameters.(1988-01-01) Nanda, P K; Sharma, M MItem Acute exacerbations of chronic obstructive pulmonary disease causes and impacts.(2014-04) Chhabra, Sunil K; Dash, Devi JyotiAcute exacerbations of chronic obstructive pulmonary disease (AECOPD) are recognised clinically as episodes of increased breathlessness and productive cough requiring a more intensive treatment. A subset of patients with this disease is especially prone to such exacerbations. These patients are labelled as ‘frequent exacerbators’. Though yet poorly characterised in terms of host characteristics, including any genetic basis, these patients are believed to represent a distinct phenotype as they have a different natural history with a more progressive disease and a poorer prognosis than those who get exacerbations infrequently. Most exacerbations appear to be associated with infective triggers, either bacterial or viral, although ‘non-infective’ agents, such as air pollution and other irritants may also be important. Susceptibility to exacerbations is determined by multiple factors. Several risk factors have been identified, some of which are modifiable. Chronic obstructive pulmonary disease (COPD) exacerbations are major drivers of health status and patient-centered outcomes, and are a major reason for health care utilisation including hospitalisations and intensive care admissions. These are associated with considerable morbidity and mortality, both immediate and long-term. These episodes have a negative impact on the patient and the disease including high economic burden, increased mortality, worsening of health status, limitation of activity, and aggravation of comorbidities including cardiovascular disease, osteoporosis and neuro-psychiatric complications. Exacerbations also increase the rate of progression of disease, increasing the annual decline in lung function and leading to a poorer prognosis. Evaluation of risk of exacerbations is now included as a major component of the initial assessment of a patient with COPD in addition to the traditionally used lung function parameter, forced expiratory volume in one second (FEV1). Decreasing the risk of exacerbations and their prevention is a major therapeutic goal of management in COPD.Item Acute exposure to toluene diisocyanate (TDI): report of a case and review of literature.(1984-10-01) Kalra, S; Malik, S K; Behera, DItem Acute fulminating pulmonary oedema following relief of airway obstruction.(1983-04-01) Dash, H H; Cheriyan, A F; Singla, RItem Acute isoniazid poisoning: presenting as seizure.(1997-10-09) Chongtham, D S; Ram, T; Jain, SA case of young female who was brought in status epilepticus with history of isoniazid poisoning is discussed. Early institution of treatment with pyridoxine saves the patient's life.