Central Retinal Vein Occlusion as a Presenting Feature Caused by Hypertensive Crisis Secondary to IgA Nephropathy

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dc.contributor.authorTekade, Pen_US
dc.contributor.authorBulchandani, Pen_US
dc.contributor.authorWairagade, N.en_US
dc.date.accessioned2023-07-14T07:02:36Z
dc.date.available2023-07-14T07:02:36Z
dc.date.issued2022-07
dc.description.abstractWe present a case of Ischemic Central retinal vein occlusion (CRVO) caused by hypertension secondary to renal failure in IgA nephropathy. A 17 year old male came with chief complaints of sudden painless diminution of vision in RE since 15 days. On examination the Right eye showed multiple superficial retinal haemorrhages in all 4 quadrents with dilated veins and cystoid macular edema suggestive of CRVO and Left eye showed superficial flame shaped haemorrhage suggestive of grade 3 hypertensive changes.en_US
dc.identifier.affiliationsVitreoretinal Services,Mahatme Eye Bank and Eye Hospital, Nagpur, Maharashtra-440025, Indiaen_US
dc.identifier.affiliationsFellow in Medical Retina and Cataract Mahatme Eye Bank and Eye Hospital, Nagpur,Maharashtra-440025, Indiaen_US
dc.identifier.affiliationsMedical Superintendent, Sr. Cornea Consultant Mahatme Eye Bank and Eye Hospital, Nagpur, Maharashtra-440025, India.en_US
dc.identifier.citationTekade P, Bulchandani P, Wairagade N.. Central Retinal Vein Occlusion as a Presenting Feature Caused by Hypertensive Crisis Secondary to IgA Nephropathy. Ophthalmology Research: An International Journal. 2022 Jul; 17(1): 8-11en_US
dc.identifier.issn2321–7227
dc.identifier.placeIndiaen_US
dc.identifier.urihttps://imsear.searo.who.int/handle/123456789/218432
dc.languageenen_US
dc.publisherSciencedomain Internationalen_US
dc.relation.issuenumber1en_US
dc.relation.volume17en_US
dc.source.urihttps://doi.org/10.9734/or/2022/v17i130246en_US
dc.subjectImmunoglobulin a nephropathyen_US
dc.subjectcentral retinal vein occlusionen_US
dc.subjectiron deficiency anemiaen_US
dc.titleCentral Retinal Vein Occlusion as a Presenting Feature Caused by Hypertensive Crisis Secondary to IgA Nephropathyen_US
dc.typeJournal Articleen_US
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