Browsing by Author "Wahi, P L"
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Item Abnormalities of haemostasis associated with congenital heart disease.(1977-01-01) Gupta, B; Jindal, S K; Mohanty, D; Das, K C; Bidwai, P S; Wahi, P LItem Acute aortic regurgitation.(1979-10-01) Mittal, S R; Wahi, P LItem Adverse reactions to sudden stoppage of drugs.(1986-03-01) Wahi, S; Wahi, P LItem Aluminium phosphide poisoning: haemodynamic observations.(1991-05-01) Kalra, G S; Anand, I S; Jit, I; Bushnurmath, B; Wahi, P LHaemodynamic measurements were made on 25 patients with aluminium phosphide poisoning. There was severe hypotension (mean arterial pressure 62.7 +/- 19.3 mmHg) and reduction in cardiac output (2.13 +/- 0.9 lit/min/m2). However, the systemic vascular resistance was only moderately elevated (2050 +/- 732 dynes/m2). The right atrial pressure was increased but the pulmonary artery and pulmonary capillary wedge pressures were normal, even in patients with pulmonary oedema. Thirteen patients (52%) who died had a lower cardiac output (p less than 0.05). Saline infusion significantly improved haemodynamics in some patients. Its judicious use early in the course of treatment may be beneficial.Item Angiocardiography in constrictive pericarditis: a study in fifty patients.(1981-11-01) Kaul, U; Anand, I S; Gupta, C D; Bidwai, P S; Wahi, P LItem Antiarrhythmic activity of racemic propranolol and its optical isomers in ouabain-induced cardiac arrhythmias in rhesus monkeys.(1975-12-01) Narula, S P; Sharma, P L; Wahi, P LItem Antimyocardial antibodies in the diagnosis and prognosis of coronary heart disease.(1977-03-01) Khahra, S; Jindal, S K; Sehgal, S; Wahi, P LItem Bone changes in congenital cyanotic heart disease and their correlation with erythrokinetic data.(1977-12-01) Jindal, S K; Mohanty, D; Das, K C; Sodhi, J S; Bidwai, P S; Wahi, P LItem Brady tachy arrhythmias in Wolff Parkinson White syndrome treated with permanent transvenous pacing and amiodrone hydrochloride.(1981-03-01) Talwar, K K; Sapru, R P; Wahi, P LItem Brain abscess in cyanotic congenital heart disease.(1989-05-01) Chakraborty, R N; Bidwai, P S; Kak, V K; Banarjee, A K; Khattri, H N; Sapru, R P; Walia, B N; Kumar, L; Suri, S; Wahi, P LTwenty-eight patients of cyanotic congenital heart disease (CHD) complicated with brain abscess were reviewed. There were 22 males and 6 females with a mean age of 9.1 +/- 5.5 years. Tetralogy of Fallot was the commonest cyanotic CHD observed. Transposition of great arteries (PS), tricuspid atresia with VSD, PS and double outlet right ventricle with VSD comprised 25% of the cardiac lesions. Febrile illness was the commonest mode of presentation (42.86%). Frontal lobe was the commonest site of abscess localization (37.5%) followed by parietal lobe (32.5%). Multiple abscess were seen in 32.14% and in 35.7% the pus was sterile on culture. Twelve patients died (mortality -42.8%), and autopsy reports were available in 6. Infective endocarditis was suspected in 7 on clinical grounds, while at autopsy, out of 6 only 2 had evidence of right-sided endocarditis. There was no correlation of mortality with age, sex, type of micro-organism, site of abscess localization and the nature of heart disease. Multiple abscesses, features of raised intracranial tension and associated meningitis/ventriculitis predicted a grim outcome.Item Captopril in hypertension a multicentric study.(1987-01-01) Kapur, A; Udwadia, B P; el-Messidi, M; Bhatia, M L; Dave, B T; Pinto, I J; Sharma, P L; Wahi, P LItem Carcinoid heart disease (a brief review of literature and a case report).(1972-07-01) Mahajan, V K; Jain, A C; Wahi, P LItem Cardiac changes in myopathy. A ballistocardiographic study.(1970-07-01) Wahi, P L; Akhtar, MItem Cardiac pacing.(1988-02-01) Wander, G S; Babu, R; Khattri, H N; Wahi, P L; Bidwai, P SItem Cardiomyopathy in Northern India: a clinico-pathological study.(1977-01-01) Babu, S K; Khatri, H N; Datta, B N; Bidwai, P S; Sapru, R P; Wahi, P LItem Cardiovascular autonomic functions in leprosy.(1982-03-01) Sahasranam, K V; Anand, I S; Kumar, B; Kaur, S; Wahi, P LItem Catecholamine-induced experimental cardiomyopathy--a histopathological, histochemical and ultrastructural study.(1989-09-01) Khullar, M; Datta, B N; Wahi, P L; Chakravarti, R NAn experimental model of myocardiopathy was induced in rhesus monkeys following noradrenaline (NA) infusion (20 ug/kg body wt/minute), for a period of 2 hours daily for three consecutive days. The animals were sacrificed after two hours (acute phase), forty-eight hours (sub-acute phase) and twenty-one days (chronic phase). Focal depletion of succinic dehydrogenase, increase in adenosine triphosphatase, acid phosphatase and appearance of large fat droplets in myocardial muscle was noted in the acute phase. Histopathological examination revealed focal edema, opacity and fuchsinorrhagia of the muscle fibres distributed in both the ventricles. Myofibrillar degeneration, myocytolysis and vacuolization with aggregation of lymphomononuclear cells were the significant features in the acute phase. During sub-acute and chronic phases, these features became less prominent and reparative changes with proliferation of fibroblasts became more marked. By the twenty-first day, irregular, focal scars replaced the necrosed myocardium. Ultrastructurally, heart muscle showed myofibrillar disorganisation, distortion of Z and A bands, dilatation of sarcoplasmic reticulum and swelling and rupture of mitochondria. Altered membrane permeability was evidenced by the presence of reaction products of horseradish peroxidase within the cardiac cells. In the reparative phase, however, myocytolytic changes regressed and collagen deposition was the prominent feature. This experimental study has several histological features simulating human cases of myocardial infarction without coronary occlusion.Item Circulating immune complexes in rheumatic heart disease.(1982-09-01) Anand, I S; Ganguly, N K; Bidwai, P S; Chugh, K S; Wahi, P LItem Clinical pattern of inferior vena cava obstruction.(1981-02-01) Chawla, A K; Jindal, S K; Singh, J D; Sodhi, J S; Pathak, I C; Wahi, P LItem A comparative double-blind adjustable dose clinical trial of penbutolol and propranolol in angina pectoris.(1977-06-01) Bhirmanandan, C V; Chadha, J S; Sharma, P L; Wahi, P L