Browsing by Author "Saini, A S"
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Item Acute effects of neurogenic stress on urinary electrolyte excretion.(1987-07-01) Marya, R K; Sood, S; Lal, H; Malik, V; Saini, A SPlasma Cortisol and urinary excretion of water, sodium, potassium, calcium and magnesium have been studied in the rat after application of 2 types of neurogenic stress:--(a) tight rubber band tourniquet and (b) electric shock. Plasma cortisol levels increased significantly after application of either type of stress. During both type of stress, there was statistically significant increase in the urinary excretion of water, sodium and calcium but not of potassium and magnesium. Urinary calcium/magnesium ratio was also significantly elevated. The results suggest that stress may be one of the factors involved in the genesis of urolithiasis.Item Adenosine deaminase activity in leprosy (a preliminary study).(1988-01-01) Chaudhary, S D; Gupta, V; Saini, A S; Singh, V; Lal, HAdenosine deaminase (ADA) activity was studied in 25 patients having different types of leprosy and 25 healthy volunteer as control. There was definite rise of ADA activity in BL (72.9 +/- 6.85), LL (56.7 +/- 3.35) and BT (39.1 +/- 8.28) which was statistically significant when compared to ADA activity in healthy control (9.7 +/- 0.53).Item Adenosine deaminase activity in pleural fluids--a diagnostic aid in tuberculous pleural effusion.(1985-04-01) Raj, B; Chopra, R K; Lal, H; Saini, A S; Singh, V; Kumar, P; Bihari, K; Chawla, R KItem Adrenal cortical function in marasmus.(1977-09-01) Tayal, J P; Saini, A S; Sood, S C; Dhatt, P S; Mehta, H C; Singh, HItem Adrenocortical involvement in aluminium phosphide poisoning.(1989-08-01) Chugh, S N; Ram, S; Sharma, A; Arora, B B; Saini, A S; Malhotra, K CThe effect of aluminium phosphide (AlP) which is a systemic poison on the adrenal cortex was studied in 30 patients of AlP poisoning. A significant rise in the plasma cortisol level (greater than 1048 nmol/l) was observed in the twenty patients. Mortality was 50 per cent. Autopsy study could be undertaken only in 10 patients. Histopathology showed mild to moderate changes. In the rest (10 patients), the adrenal cortex was critically involved and the cortisol level failed to rise beyond normal levels (less than 690 nmol/l). The histopathology revealed severe changes (complete lipid depletion, haemorrhage, necrosis etc.) and all these patients died. In the critically ill patients, the cortisol levels remained low because of severe adreno-cortical involvement. The changes in the adrenal cortex could be due to shock or to cellular toxic effect of phosphine. The histopathological changes in various viscera showed congestion, edema and cellular infiltration. In the heart, there were patchy areas of necrosis, while the liver showed fatty changes and the lungs showed, in addition areas of gray/red hepatization. There was no adrenal apoplexy or extensive haemorrhage that could explain shock in these patients. Cardiogenic shock could not be confirmed due to lack of facilities for haemodynamic monitoring, but there was histopathological evidence in support of cardiovascular shock.Item Alterations in blood free alpha-amino nitrogen and protein levels in chronic renal failure.(1979-04-01) Lal, H; Gupta, R R; Saini, A S; Parkash, CItem Biochemical assessment of PEM.(1985-01-01) Grover, A K; Singh, H; Mehta, H C; Saini, A S; Dhatt, P S; Sood, S CItem Blood and urinary alpha ketoacids in normal adults and their levels in relation to the menstrual cycle in females.(1971-05-01) Mahajan, K K; Bajaj, V R; Saini, A S; Mahajan, S KItem Blood and urinary ketoacids in Indian childhood cirrhosis.(1967-07-01) Dhatt, P S; Gupta, H L; Saini, A SItem Carboxy-haemoglobin levels following cigarette and bidi tobacco smoking--a comparative study.(1985-06-01) Gupta, S G; Gupta, M S; Moga, R L; Saini, A SItem Cation and water transport during maturation of cortical cataract.(1988-01-01) Khurana, A K; Lal, H; Saini, A S; Chauhan, B S; Walia, B KItem Chromatographic separation of sugars from untreated urine on cellulose thin layers.(1975-04-01) Mehta, H C; Saini, A SItem Disaccharide intolerance & ICC. VII. Effect of steroid therapy on lactosuria.(1987-02-01) Chugh, J C; Dhatt, P S; Singh, H; Saini, A S; Mehta, H CItem Disaccharide intolerance and Indian childhood cirrhosis. V. Effect of lactose administration on melituria in parents of ICC patients.(1983-07-01) Dhatt, P S; Sabharwal, H S; Singh, H; Saini, A S; Mehta, H C; Niharika,Item Disaccharide intolerance and Indian childhood cirrhosis: a possible etiological factor.(1968-01-01) Dhatt, P S; Gupta, H L; Saini, A SItem Disaccharide intolerence and Indian childhood cirrhosis. IV. Relationship of melituria to lactose administration.(1982-07-01) Dhatt, P S; Aggarwal, R K; Saini, A S; Singh, H; Sood, S C; Mehta, H CItem Disaccharide intolerence and Indian childhood cirrhosis. VI. The nature of excreted disaccharide.(1986-03-01) Chugh, J C; Singh, H; Saini, A S; Dhatt, P S; Mehta, H CItem Effect of acute environmental heat stress on urinary water and electrolyte excretion in the rat.(1988-04-01) Marya, R K; Sood, S; Lal, H; Sharma, A; Saini, A SPlasma cortisol and urinary excretion of water, sodium, potassium, calcium and magnesium have been studied in the rat after application of heat stress. There was a significant increase in plasma cortisol level after exposure to heat. During heat stress complete cessation of urine formation was observed. In the next 30 min there was statistically significant increase in the urinary excretion of water, sodium and calcium but not of potassium and magnesium. Urinary calcium/magnesium ratio was also significantly elevated. The increase in urinary water and electrolyte excretion seemed to be mediated through prostaglandins since it could be abolished by administration of indomethacin prior to the application of heat stress. On the basis of these results, the possible role of heat stress in the genesis of urolithiasis has been discussed.Item Effect of dietary restriction with and without leucine supplementation on hepatic protein status in rats.(1988-01-01) Chugh, K; Lal, H; Shanker, V; Saini, A SIn the present study effect of dietary restriction with and without leucine supplementation was observed on body and liver weights, and liver protein status, in adult rats. Animals were fed on two diets ad lib or were on 50 per cent and 25 per cent intakes. Dietary restriction resulted in loss of body and liver weights, hepatic protein, free-alpha-amino nitrogen and RNA contents and liver cell size (liver weight/DNA ratio). When compared with the control group, the decrease in these parameters was more in the dietary restricted leucine supplemented group. However, hepatic DNA content was not changed with the change in dietary regimen. The results suggest that leucine supplementation with dietary restriction may be more harmful for the animal than dietary restriction alone.Item Effect of prolonged starvation and refeeding on fuel metabolism in rats.(1985-04-01) Sharma, M; Lal, H; Saini, A SBody and liver weights, Liver lipids, glycogen, aspartate aminotransferase (EC 2.6.1.1), alanine aminotransferase (EC 2.6.1.2) and blood glucose levels were determined in starved and starved-refed rats. Decrease in body and liver weights was rapid during the initial stage of starvation and slowed down thereafter. Water was the major liver constituent lost in early fast. Following 10 days of starvation, body weight was reduced by nearly 20%, liver weight 43%, liver glycogen 93% and blood glucose 34%. Liver lipids and the activities of the two transaminases however, were increased by about 30-50%. On refeeding body weight and its water content increased and became nearly double of the initial fasting value on day 2. Blood glucose, liver glycogen, liver lipids and transaminases were significantly altered and got normalised within 5-8 days.
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