Browsing by Author "Nayak, N C"
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Item A 20-year-old man with recurrent abdominal pain and vomiting since the age of 5 years.(2008-11) Wig, J D; Nayak, N CItem Acute myeloid leukaemia terminating in histiocytic medullary reticulosis.(1982-07-01) Juneja, S K; Nayak, N C; Raina, V; Chopra, PItem Aflatoxin B1 induced hepatocarcinogenesis in neonatal rats.(1992-03-01) Mathur, M; Rizvi, T A; Nayak, N CRole of cell replication on aflatoxin B1 (AFB1) induced hepatocarcinogenesis was investigated in neonatal rats showing persistence of cell replication in the liver for 21 days of post natal life. Adult (8-10 weeks old) rats displaying no hepatocytic proliferation served as controls. Three doses of AFB1 were administered to both the groups at intervals of 48 hr with the doses starting on 10th day of age in the neonatal group. Appearance of phenotypically altered preneoplastic hepatocytes was quantitated in both the groups. A significantly higher incidence of preneoplastic foci was recorded in neonatal rats as compared to adult animals. The results suggest that presence of cell replication in neonatal rats at the time of AFB1 administration enhances the process of hepatocarcinogenesis.Item Antithyroid antibodies & thyroiditis--a correlative study in adenomatous goitre & adenoma thyroid.(1984-11-01) Dali, S M; Chopra, P; Kapur, M M; Nayak, N CItem Biochemical & immunochemical analysis of liver specific protein.(1985-01-01) Panda, S K; Kar, S K; Nayak, N CItem Carbon tetrachloride toxicity: failure of promethazine to prevent in vivo liver injury.(1980-10-01) Nayak, N C; Chopra, PItem Cardiomyopathy. Clinical, haemodynamic and pathological features.(1973-09-01) Bhatia, M L; Wasir, H S; Nayak, N C; Roy, S BItem A case of abscess in a cirrhotic liver rupturing into the right subdiaphragmatic and perinephric spaces and involving the right kidney.(1966-08-01) Ahuja, M M; Nayak, N C; Bhargava, SItem Cassia occidentalis poisoning as the probable cause of hepatomyoencephalopathy in children in western Uttar Pradesh.(2007-06-21) Vashishtha, V M; Kumar, Amod; John, T Jacob; Nayak, N CBACKGROUND & OBJECTIVE: Recurrent annual outbreaks of acute encephalopathy illness affecting young children have been reported for several years in many districts of western Uttar Pradesh (UP). Our earlier investigations over three consecutive years (2002-2005) proved that these outbreaks were due to a fatal multi-system disease (hepatomyoencephalopathy syndrome) probably caused by some phytotoxin and not due to viral encephalitis as believed so far. We conducted a case-control study to investigate the risk, if any, from various environmental factors and also to identify the putative toxic plant responsible for development of this syndrome. METHODS: Eighteen cases with acute hepatomyoencephalopathy syndrome admitted in 2005 in a secondary care paediatric hospital of Bijnor district of western UP were included in the study. Three age-matched controls were selected for each case. A semi-structured questionnaire was developed and applied to all 18 cases and 54 controls. All interviews were conducted within one week of discharge or death of each case. Quantitative data were analyzed using the relevant established statistical tests. RESULTS: Parents of 8 (44.4%) cases gave a definite history of their children eating beans of Cassia occidentalis weed before falling ill, compared with 3 (5.6% controls), the odds ratio being 12.9 (95% CI 2.6-88.8, P<0.001). History of pica was the other associated factor with the disease, odds ratio 5.20 (95% CI 1.4-19.5, P<0.01). No other factor was found significantly associated with the disease. INTERPRETATION & CONCLUSION: Consumption of C. occidentalis beans probably caused these outbreaks, described earlier as hepatomyoencephalopathy syndrome. Public education has the potential to prevent future outbreaks.Item Cassia occidentalis poisoning causes fatal coma in children in western Uttar Pradesh.(2007-07-09) Vashishtha, Vipin M; Kumar, Amod; John, T Jacob; Nayak, N CWe investigated cases of the annual seasonal outbreaks of acute hepato-myo-encephalopathy in young children in western Uttar Pradesh for causal association with Cassia occidentalis poisoning, by a prospective survey in 2006. During September-October homes of 10 consecutive cases were visited and history of eating Cassia beans was obtained in all. Nine children died within 4-5 days. There appears to be an etiological association between consumption of Cassia occidentalis beans and acute hepato-myo-encephalopathy.Item Cellular evolution of hepatic cancer in rats.(1983-07-01) Manga, A; Chopra, P; Nayak, N CItem Clinical and histological evaluation of solitary thyroid nodules.(1972-01-01) Bhatia, H K; Ahuja, M M; Kumar, V; Kapoor, M M; Nayak, N CItem Combined effect of small doses of dimethylnitrosamine and prior liver injury on the induction of kidney tumour in the rat.(1977-07-01) Chopra, P; Dhar, A; Nayak, N CItem Commercial terylene cloth for the repair of abdominal wall defects.(1972-03-01) Kapur, B M; Rao, B N; Nayak, N C; Mohanty, SItem Comparative data on the detection of hepatitis-B surface antigen by six centres.(1981-02-01) Baxi, A J; Bhatia, H M; Dutta, R N; John, T J; Joshi, S H; Kulkarni, K V; Nayak, N C; Pal, S R; Vyas, G NItem Demonstration of hepatitis B virus surface component in human hepatocellular cancer cells.(1979-01-01) Nayak, N C; Sachdeva, R; Dhar, A; Seth, H NItem Dynamics of cellular and humoral immunity to liver cell membrane antigens in acute viral hepatitis.(1981-07-01) Pranava, M; Nath, I; Nayak, N C; Joshi, Y K; Tandon, B NItem Effect of prolonged administration of noradrenaline, with and without heparinization in irreversible haemorrhagic shock in dogs.(1968-02-01) Balkrishna Rao, B N; Upadhyaya, P; Nayak, N C; Kalhan, J NItem Etiologic spectrum of acute sporadic viral hepatitis in children in India.(1989-04-01) Panda, S K; Datta, R; Gupta, A; Kamat, R S; Madangopalan, N; Bhan, M K; Rath, B; Guha, D K; Nayak, N CThe relative magnitude by hepatitis A virus (HAV), hepatitis B virus (HBV) and hepatitis Non-A, Non-B virus (HNANBV) was determined in 496 children from three different parts of India suffering from acute viral hepatitis by tests for specific IgM class anti-HAV and anti-HBV antibodies in the serum. HAV, HBV and NANB infections accounted for 55.8 per cent, 20.2 per cent and 23.2 per cent of cases respectively. Hepatitis A largely (59.5%) affected younger children of 1-5 yr. Nearly a third of children affected by NANB hepatitis were additionally positive for HBsAg. The proportions of HAV and HBV infected cases respectively decreased and increased with increasing age whereas the incidence of HNANBV infection remained almost constant throughout childhood. Acute NANB hepatitis, a major health problem in the adults of India is also common throughout childhood. This study suggests that this infection does not impart long lasting protective immunity.Item Evaluation of low molecular weight dextran in canine endotoxin shock.(1972-03-01) Kapur, B M; Rao, B N; Nayak, N C; Sarangee, R K
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