Browsing by Author "Misra, N P"
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Item Abbreviated rapid onset stress testing for exposure of ventricular arrhythmias in post-hospital phase of myocardial infarction.(1983-11-01) Manoria, P C; Misra, N P; Bhargava, R KItem Aetiopathogenesis of hemiplegia in patients below 40 years--a clinical and angiographic study.(1980-03-01) Misra, N P; Sharma, K N; Varma, YItem Atenolol in hypertension.(1984-10-01) Misra, N P; Bahadur, M MItem Bhopal tragedy--a year later.(1986-04-01) Misra, N PItem Bronchoalveolar lavage study in victims of toxic gas leak at Bhopal.(1989-12-01) Vijayan, V K; Pandey, V P; Sankaran, K; Mehrotra, Y; Darbari, B S; Misra, N PBronchoalveolar lavage using flexible fibreoptic bronchoscope was carried out in 50 patients 1-2 1/2 yr after exposure to the 'toxic gas' at Bhopal. Thirty six patients in the analysis were categorised into 3 groups (viz., mild, moderate and severe), depending upon the severity of exposure. There was an increase in cellularity in the lower respiratory tract (alveolitis) of the severely exposed patients (in both smokers and non-smokers), compared to normals (P less than 0.05). The increase in cellularity in severely exposed non-smokers was due to abnormal accumulation of macrophages (P less than 0.01), and in severely exposed smokers, to macrophages (P less than 0.01) and neutrophils (P less than 0.05). Mild and moderately exposed patients did not show significant change in cellularity in lower respiratory tract, compared to normal individuals (P greater than 0.2). There was a trend towards increasing cellularity, as the severity increased (P less than 0.0001) and higher numbers of total cells were seen in severely exposed smokers, suggesting that smoking is a risk factor. It appears, therefore, that subjects severely exposed to the toxic gas at Bhopal may have a subclinical alveolitis characterised by accumulation and possibly activation of macrophages in the lower respiratory tract. Smokers, who were exposed to the gas had in addition, accumulation of neutrophils.Item Bumetanide in congestive cardiac failure.(1983-12-01) Misra, N P; Jain, S C; Maheshwari, JItem Captopril in congestive cardiac failure.(1986-11-01) Misra, N P; Manoria, P C; Trivedi, H H; Mehrotra, Y; Pandey, PItem Cardiac arrhythmias after administration of digoxin through intravenous, intra-cerebroventricular and combined routes. An experimental study.(1981-03-01) Misra, N P; Manoria, P C; Bhargava, R K; Bhagwat, A W; Kohli, NItem Clinical profile of gas leak victims in acute phase after Bhopal episode.(1987-01-01) Misra, N P; Pathak, R; Gaur, K J; Jain, S C; Yesikar, S S; Manoria, P C; Sharma, K N; Tripathi, B M; Asthana, B S; Trivedi, H HItem Clinical status of tricyclic antidepressants in peptic ulcer.(1986-05-01) Tripathi, B M; Misra, N P; Rao, SItem Clinico-pathological and experimental study of epidemic dropsy.(1984-11-01) Misra, N P; Varma, P; Jain, S C; Saify, A Q; Bhagwat, R RItem A comparative study of flurbiprofen and piroxicam in osteoarthritis.(1992-10-01) Misra, N PIn this single-blind, multiple-dose study the efficacy and tolerability of flurbiprofen was compared with that of piroxicam in 60 adult patients suffering from osteoarthritis of the knee. The patients were randomly allocated to receive either flurbiprofen 100 mg twice daily or piroxicam 20 mg once daily for a period of four weeks. Clinical assessments w.r.t. pain, tenderness, stiffness, swelling and general activity of patient were carried out prior to initiation of trial therapy and thereafter at weekly intervals for four weeks. The findings were graded. Though significant improvements as compared to baseline data occurred in both the treatment groups, flurbiprofen was found to be superior to piroxicam in improving pain on movement and at rest (p < 0.05). The incidence of side effects was less in the group receiving flurbiprofen (6% compared to 47% observed with piroxicam).Item Congenital eventration of the left dome of the diaphragm.(1982-07-01) Misra, N P; Manoria, P C; Sharma, P NItem Convenient new dosage schedule of tetracycline.(1979-08-01) Misra, N P; Gaur, K JItem Diagnostic value of ascitic fluid examination.(1966-01-01) Jain, S C; Misra, S M; Misra, N P; Tandon, P LItem Double blind randomised study of tripotassium-di-citrato-bismuthate in the treatment of peptic ulcer.(1984-11-01) Tripathi, B M; Misra, N P; Dube, SItem Early observations on lung function studies in symptomatic "gas" exposed population of Bhopal.(1987-01-01) Bhargava, D K; Verma, A; Batni, G; Misra, N P; Tiwari, U C; Vijayan, V K; Jain, S KItem Effect of beta adrenergic blockade on gastric secretion.(1978-11-01) Tripathi, B M; Sharma, R K; Misra, N PItem Electrocardiographic changes in the first month of life.(1981-01-01) Misra, N P; Manoria, P C; Bhargava, R K; Trivedi, H H; Gulati, B; Kanojiya, R SItem Enalapril in hypertension.(1992-07-01) Misra, N P; Verma, Y; Shah, J YTwenty five patients of mild to moderate uncomplicated essential hypertension and five with severe hypertension were treated with long acting converting enzyme inhibitor enalapril for six weeks. Diuretic was added in those patients who did not respond satisfactorily. Twenty one patients of mild to moderate hypertension had their diastolic blood pressure controlled at the end of the study; fifteen with enalapril alone and six with the help of diuretic. Remaining four showed a relative fall but not to level below 150/90 mm Hg. Only one patient with severe hypertension showed fall to normal levels. Four showed a relative fall but not to the normal level even with the addition of a diuretic. Enalapril is an effective anti hypertensive drug in mild to moderate essential hypertension.
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